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 BOF 123

 

   

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BOF 123
AML M3 subtype, all are true except
1-t(15,17) confers a good prognosis
2-the genetic defect lies in RARA gene
3-treatment with ATRA enhances further differentiation of the promyeloblasts from its differentiation block site
4-almost always of a hypogranular type
5-evidence of DIC is present up to 80% at the time of diagnosis

 

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